Skin

This necrosis and/or tecidual isquemia, are presented in characteristic points of pressure for the drawn out corporal positioning, of which they show to the figures and the B. According to ROCK, 2006, the localizations most frequent are: isquitica (24%), sacrococcgea (23%), trocantrica (15%), and calcnea 8 (%). Other localizations include malolos lateral (7%), elbows (3%), occipital region (1%), and region to escapular. FISIOPATOLOGIA the skin possesss an abundant sanguineous suppliment that supplies to oxygen to all its layers. When this sanguineous suppliment is interrupted for more than 2 or 3 hours, the skin starts to die, starting for its more external layer (the epidermis). Having as common cause the pressure stops of reduction of the sanguineous flow for the skin. Click Somatic Experiencing to learn more.

Some factors are implied in the cutaneous injury between them are distinguished it pressure, forces boost glide, friction and humidity. The normal skin can resist the pressures between 200 the 600 mmHg for up to 11 the 16 hours. In the populations with increased risk, ischemic ulcers if develop when an area suffers pressure from 500 mmHg for 2 150 hours or mmHg for 10 hours. The increased pressure induces the tecidual isquemia, followed of one eritema reacional. A prolongation of the isquemia cause plasma extravasation for the interstice and can appear hemorrhage in the place. The skin can be more resistant than the fabric subcutaneous and muscular, that the appearance of amolecida tumorao allows, that can be infectar in bacteremia case (endogenous infection). The forces boost glide that happen in the patients acamados with the headboard of the high stream bed that slides in direction to the foot of the bed, strangle the subcutaneous vases and unglue the skin, increasing the tumorao. The local friction and humidity finally provoke the erosion of the skin. The ulcers that involve only the epidermis or the extension of derme take 2 days the weeks stop to heal.